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By N M Hooper; Humana Press

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1993) Familial Alzheimer’s disease: a pedigree with a missense mutation in the amyloid precursor protein (amyloid precursor protein 717 valine-glycine). Brain 116, 309–324. 45. , and Benson, M. (1994) Clinical characteristics in a kindred with early onset Alzheimer’s disease and their linkage to a G-T change at position 2149 of the amyloid precursor protein gene. Neurology 44, 105–111. 46. , et al. (1992) A new mutation in APP demonstrates that the pathogenic mutations for probable Alzheimer’s disease frame the β-amyloid sequence.

18, 69–71. 42 Brindle and St. George-Hyslop 83. , et al. (1998) Absence of association between Alzheimer disease and the –491 regulatory region polymorphism of APOE. Neurosci. Letts. 250, 189–192. 84. , et al. (1996) Genetic association of very low density lipoprotein (VLDL) receptor gene with sporadic Alzheimer’s disease. Nature Genet. 11, 207–209. 85. , Gauntlett, A. , Cai, X. , et al. (1997) No association between the very-low density lipoprotein receptor gene and late-onset Alzheimer’s disease, nor interaction with apolipoprotein E gene in population based and clinic samples.

D. (1997) Synergy between the genes for butyrylcholinesterase K variant and apolipoprotein E4 in late onset confirmed Alzheimer’s Disease. Hum. Mol. Genet. 6, 1933–1936. 107. , Masters, C. , and Heuser, I. (1998) Butyrylcholinesterase K variant (BCHE-K): association with late-onset Alzheimer’s disease. Neurobiol. Aging 19, S69. 108. , et al. (1998) Analysis of the butyrylcholinesterase gene and nearby chromosome 3 markers in Alzheimer’s disease and aging. Hum. Mol. Genet. 7, 933–935. 109. Singleton, A.

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